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Jacqueline A. Hobbs, M.D., Ph.D.
Assistant Professor
Dr. Hobbs graduated summa cum laude with a B.A. in Biology from DePauw University in Greencastle, Indiana. She then attended Indiana University School of Medicine from which she graduated from the M.D./Ph.D. program with her graduate work in microbiology and immunology. Her thesis work focused on the mechanism of cell death caused by the rabies-related virus, vesicular stomatitis virus. Dr. Hobbs completed her first year of Psychiatry training at Mount Sinai School of Medicine in New York City. She then completed her second and third years of training at Indiana University School of Medicine in Indianapolis, Indiana before moving to the University of Florida where she completed her final year of Psychiatry residency training. Upon completion of her Psychiatry residency, she joined the faculty of the University of Florida College of Medicine as an assistant professor. Dr. Hobbs received funding from the Stanley Medical Research Institute while still a resident to aid her in her studies of viral etiologies of schizophrenia. She continues this work as a faculty member in the Department of Psychiatry.
Clinical Interests
Dr. Hobbs' clinical interests include schizophrenia and other psychoses, bipolar disorder, and women’s mental health.
Research and Scholarly Interests
Dr. Hobbs' research interests include neurovirology, animal models of virus infection in the developing brain, viral/infectious etiologies of schizophrenia and bipolar disorder as well as other mental illnesses.
Educational Interests and Accomplishments
Dr. Hobbs serves as an attending psychiatrist in the adult outpatient clinic and the on-call service where she regularly supervises and teaches residents during their second year of training. She also values the one-on-one teaching of students in the laboratory. She is currently mentoring two medical students in their first and second years of medical school in her laboratory. Dr. Hobbs lectures on general psychiatric topics to physician-assistant students.
Leadership and Service
Dr. Hobbs regularly interviews perspective residency and faculty candidates for the Department of Psychiatry.
Publications
Hobbs, J.A. 2006. Detection of adeno-associated virus 2 and parvovirus B19 in the human dorsolateral prefrontal cortex. Journal of NeuroVirology, 12:1-10.
Hobbs, J.A. and A. Shekhar. 2003. Developmental aspects of panic and related anxiety disorders. Neuroembryology 2:72-80.
Hobbs, J.A., G. Hommel-Berrey, and Z. Brahmi. 2003. Requirement of caspase-3 for efficient apoptosis induction and caspase-7 activation but not viral replication or cell rounding in cells infected with vesicular stomatitis virus. Human Immunol. 64:82-92.
Hobbs, J.A., S. Cho, T.J. Roberts, V. Sriram, J. Zhang, M. Xu, and R. Brutkiewicz. 2001. Selective loss of natural killer T cells by apoptosis following infection with lymphocytic choriomeningitis virus. J. Virol. 75:10746-10754.
Hobbs, J.A., R.H. Schloemer, G. Hommel-Berrey, and Z. Brahmi. 2001. Caspase-3-like proteases are activated by infection but are not required for replication of vesicular stomatitis virus. Virus Research 80:53-65.
Spence, P.M., V. Sriram, L. Van Kaer, J.A. Hobbs, and R.R. Brutkiewicz. 2001. Generation of cellular immunity to lymphocytic choriomeningitis virus is independent of Cd1d1 expression. Immunology 104:168-174.
Hobbs, J.A. and Z. Brahmi. 1999. Vesicular stomatitis virus infection: requirement of caspase-3 activation for the efficient induction of apoptosis but not viral replication. IBASM News, September, 1999.
Hobbs, J.A. 1999. Vesicular stomatitis virus infection: requirement of caspase-3 activation for the efficient induction of apoptosis but not viral replication. Doctoral dissertation, Indiana University School of Medicine.
N’Cho, M., J.A. Hobbs, and Z. Brahmi. 1999. Overexpression of multidrug resistance P-glycoprotein inhibits granule-mediated lytic ability and causes loss of cytoplasmic perforin in NK cells without affecting their Fas-mediated cytotoxicity. Human Immunol. 60:223-230.
Csipo, I., A.H. Montel, J.A. Hobbs, P.A. Morse, and Z. Brahmi. 1998. Effect of Fas+ and Fas- target cells on the ability of NK cells to repeatedly fragment DNA and trigger lysis via the Fas lytic pathway. Apoptosis 3:105.
Montel, A.H., M.R. Bochan, J.A. Hobbs, D.H. Lynch, and Z. Brahmi. 1995. Fas involvement in cytotoxicity mediated by human NK cells. Cell. Immunol. 166:236.
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